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    Impotence or, to give it its correct name, erectile dysfunction affects an astounding one in ten men in the UK. Until recently, the disorder was a taboo subject, but with increased publicity in the last few years, even the most reluctant men are beginning to seek medical help. For years, the only treatment option was injections into the penis. Then came Viagra, the little blue pill that works directly on smooth muscle - a type of muscle in our bodies that we can't control consciously - to allow blood to flow into the penis and cause an erection. To date, 15 million men worldwide have depended on Viagra to stop their impotence. But this summer a new drug, Uprima, was released onto the market which works on the parts of the brain that control smooth muscle contractions to give the same effect as Viagra, only quicker. Uprima targets the parts of the brain which control the movement of smooth muscle in the penis, stimulating it to relax and boosting blood flow. There are many alternatives to ED Medications to treat erectile dysfunction (ED), her are three of the best. The ways we have found to treat erectile dysfunction have evolved greatly over the past 20 years. Before the introduction of drugs such as Viagra, which are known as phosphodiesterase inhibitors (PDE5i), we were able to offer patients one of only two definitive treatments for ED: penile implants or surgical penile revascularization. The introduction of the PDE5 inhibitors introduced a new era of non-invasive treatments for erectile dysfunction. We now have five oral ED medications available: the early introductions – Viagra, Cialis, Levitra – and newer ones, Stendra, Staxyn. While these drugs can be very effective, we know they are There are a few major problems with Viagra and the other oral ED drugs. There are certain men who cannot take them at all, they produce a variety of side-effects, even in men that can take them, and most importantly, they do not treat the root cause of ED.

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    Just over one year ago here at Diabetes Flashpoints, we discussed the possibility that hundreds of thousands of people with both diabetes and kidney disease might benefit from taking the diabetes drug metformin. As we noted then, this drug has carried a “black box” warning on its label — mandated by the U. Food and Drug Administration (FDA) — ever since it became available in the United States in 1994, due to concerns about lactic acidosis. Lactic acidosis is much more common in people with impaired kidney function. This rare but extremely serious reaction was found to be an unacceptably common side effect of a drug related to metformin — phenformin — which was pulled from the U. Since metformin’s warning label is based, in part, on concerns about a different drug entirely, many researchers have tried to estimate how safe metformin is for people with diabetes whose kidney function is impaired. Last year, we noted that many researchers believe metformin is safe for people with mild to moderate kidney disease, defined as having an estimated glomerular filtration rate (e GFR) of 30–60 ml/min. And one study found that using a safety cutoff of an e GFR of 30 ml/min, nearly one million people in the United States who currently don’t take metformin because of the FDA’s labeling might be able to safely do so. The evidence, it seems, has only grown stronger in favor of metformin being more widely prescribed to people with kidney disease. As noted in a recent article at Diabetes In Control.com, the blood-glucose-lowering benefits of loosening restrictions on metformin could be enormous. One study cited in the article, published last August in the journal Diabetes Care, found that depending on how e GFR is calculated, as many as 834,000 people could be newly eligible for metformin if the eligibility threshold were set at an e GFR of 45 ml/min or higher. Metformin safe in patients with mild, moderate chronic kidney disease where can i buy celexa FDA Revises Recommendation for Metformin Use in Patients With. Metformin and the Risk for Lactic Acidosis in CKD - Medscape
     
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